Which medications are known to facilitate the shift of potassium from serum into cells?

Insulin, particularly when administered in conjunction with glucose, is highly effective in facilitating the movement of potassium from the serum (the bloodstream) into the cells. This occurs because insulin stimulates the sodium-potassium ATPase pump, which actively transports potassium into cells in exchange for sodium. In clinical practice, this mechanism is often utilized to manage hyperkalemia, a condition characterized by elevated levels of potassium in the blood. By administering insulin with glucose, the risk of hypoglycemia is mitigated while effectively lowering serum potassium levels.

Other options, while they may have some roles in electrolyte management or related processes, do not have the same direct and effective impact on shifting potassium into cells as the combination of insulin and glucose does. Corticosteroids can affect potassium levels, but their primary mechanism is different, and they are not primarily used for the rapid correction of hyperkalemia. Propranolol and aspirin do not have significant roles in potassium shifting, and calcium is typically known for managing cardiac effects of hyperkalemia rather than directly facilitating potassium movement into cells.